June 29, 2012 ~ St. Pete Beach, FL

Mitochondrial Physiology of Barth Syndrome

Linking cardiolipin remodeling to mitochondrial beta-oxidation (PDF Presentation)
Grant Hatch, PhD, University of Manitoba, Winnipeg, Canada

Modeling Barth syndrome in cardiomyocytes generated from patient-derived induced pluripotent stem cells (PDF Presentation)
William Pu, MD, Children´s Hospital of Boston, Boston, MA

The preferred acyl chain donor of the yeast tafazzin (PDF Presentation)
Anton I. de Kroon, PhD, Utrecht University, Utrecht, The Netherlands

Cardiolipin remodeling by ALCAT1 regulates dilated cardiomyopathy through oxidative stress and mitophagy (PDF Presentation)
Yuguang (Roger) Shi, PhD, Penn State University College of Medicine, Hershey, PA

Brainstorming and conference wrap-up
Richard I. Kelley, MD, PhD, Kennedy Krieger Institute Department of Pediatrics, Johns Hopkins University, Baltimore, MD, USA; Grant Hatch, PhD, University of Manitoba, Winnipeg, Canada; Michael Schlame, MD, New York University, New York, NY